Wolfson Institute for Biomedical Research


Scanning electron micrograph of the dendrites of a cerebellar Purkinje cell - Hausser Lab


Wolfson Institute for Biomedical Research

The Wolfson Institute for Biomedical Research was established at UCL in 1995 for the pursuit of excellence in translational biomedical research. Now, with its primary focus on fundamental and translational neuroscience, the WIBR is a key hub within the rich environment of the UCL neuroscience community.
Lab Research Photo


The WIBR is currently home to 14 Principal Investigators, including established senior academics as well as early-career scientists. Collectively we hold more than £25m in grants from The European Research Council, MRC, BBSRC, Wellcome and other biomedical research charities.

Teaching in lab


Researchers within the WIBR organise and contribute to several distinguised undergraduate modules within the School of Life and Medical Sciences. The Institute has around 20 current PhD students, and runs a highly rated one-year MSc degree course in Drug Design.

wood lab group photo


Contact details for WIBR staff

Contact Us (WIBR)

Contact Us

Our address and location

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MRes Drug Design Programme

The MRes Drug Design programme, with research projects hosted by labs from the Wolfson Institute for Biomedical Research offers advanced training in cutting edge neuroscience-related techniques.


WIBR History

Find out more about WIBR history

https://www.cancertools.org/tools2023 Publications

Iseppon, F., Luiz, A. P., Linley, J. E., & Wood, J. N. (2023). Pregabalin silences oxaliplatin-activated sensory neurons to relieve cold allodyniaeNeuro, ENEURO.0395-ENEU22.2022. doi:10.1523/ENEURO.0395-22.2022

Qin, Q., Wang, M., Li, H., Xu, Z. D., & Tang, Y. (2023). Editorial: The role of microglia in the pathogenesis of neurodegenerative diseasesFrontiers in Aging Neuroscience, 14, 1105896. doi:10.3389/fnagi.2022.1105896

Zhu, W., Li, W., Jiang, J., Wang, D., Mao, X., Zhang, J., . . . Li, N. (2023). Chronic salmon calcitonin exerts an antidepressant effect via modulating the p38 MAPK signaling pathwayFrontiers in Molecular Neuroscience, 16, 1071327. doi:10.3389/fnmol.2023.1071327