UK Parkinson's Disease Consortium - UKPDC
- Principal Investigators
- Research Groups
- Cell Physiology
- Clinical Neuroscience
- Clinical Studies
- Drosophila Genetics
- Molecular Biology and Biochemistry
- Molecular Neuropathology
- Neurological Biochemistry
- Neurological Signalling
- Protein Phosphorylation
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Professor John Hardy (UCL Institute of Neurology) has been awarded the $3 million Breakthrough Prize in Life Sciences for his pioneering research into the genetic causes of Alzheimer’s disease, other forms of dementia and Parkinson’s disease. More...
One of the UK Parkinson's Disease Consortium Principal Investigators, Prof John Hardy, has been awarded the 2015 Robert A. Pritzker Prize for his leadership in Parkinson's genetics research. The award was presented by Michael J. Fox at a ceremony in New York on April 15. From the Michael J. Fox Foundation website: More...
Webcast of the presentation entitled ‘Advances in Genetic Understanding of Parkinson's Disease’ given by Nicholas Wood (University College London, United Kingdom) presented at the Biochemical Society Hot Topic event, PINK1-Parkin Signalling in Parkinson’s Disease and Beyond, held in December 2014. More...
A study published in Brain, led by researchers
at UCL Institute of Neurology, has shown that genetic mutations which
cause a decrease in dopamine
production in the brain and lead to a form of childhood-onset Dystonia,
also play a role in the development of Parkinson’s disease.
The new Leonard Wolfson Experimental Neurology Centre (LWENC) has opened for clinical studies and trials
(Former Research Associate)
Mitochondrial Morphology and distribution. Apoptosis, autophagy and mitochondrial electron transport chain function in relation to Parkinson’s Disease.
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Mitochondria and quality control defects in a mouse model of Gaucher Disease-links to Parkinson's Disease.
Cellular and molecular mechanisms of mitochondrial function.
Best Pract Res Clin Endocrinol Metab
Accumulation of damaged mitochondria in neuropathic Gaucher disease, the most prevalent risk factor for Parkinson's disease, is due to defective cellular degradation machinery.
Presented at: UNSPECIFIED.
MiD49 and MiD51, new components of the mitochondrial fission machinery.
565 - 573.
The regulation of mitochondrial morphology: Intricate mechanisms and dynamic machinery.
1534 - 1545.
MiD51 and MiD49: New mediators of mammalian mitochondrial distribution.
BIOCHIMICA ET BIOPHYSICA ACTA-BIOENERGETICS.
(pp. S57 - S57).
ELSEVIER SCIENCE BV
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