UCL Institute of Ophthalmology


IoO Seminar Series

26 January 2022, 2:30 pm–5:00 pm

IoO Seminar Series banner with Seth Blackshaw

Speaker: Seth Blackshaw from the Solomon H. Snyder Department of Neuroscience, Johns Hopkins University.

Event Information

Open to

All | UCL staff | UCL students




Trudy Muggridge


14:30-15:30  Seminar and questions
15:30-16:00  Postdoc/PhD student discussion
16:00 -1700  PI roundtable discussion

About the Speaker

Seth Blackshaw

at UCL Institute of Ophthalmology

Professor Seth Blackshaw is a professor of neuroscience, neurology and ophthalmology at the Johns Hopkins University School of Medicine. Additionally, he serves as an investigator in both the High Throughput Biology Center and the Institute for Cell Engineering at Johns Hopkins. Dr Blackshaw received a B.A. in biology and an M.S. in biochemistry from the University of Chicago in 1991. He completed his PhD in neurosciences at Johns Hopkins in 1997 and subsequently conducted postdoctoral work in genetics at the Harvard University Medical School. He joined the Hopkins faculty in 2004. Dr Blackshaw has authored or co-authored more than 110 peer-reviewed publications and holds several patents and copyrights. His laboratory aims to identify genes that pattern progenitors in time and regulate their ability to proliferate and give rise to specific types of retinal cells at different stages during the course of neurogenesis.

Website: https://blackshawlab.com

Recent publications: 

  • Lyu et al. 2021. Gene regulatory networks controlling temporal patterning, neurogenesis, and cell-fate specification in mammalian retina. Cell Reports. 10.1016/j.celrep.2021.109994
  • Weir et al. 2021. Multiplexed Analysis of Retinal Gene Expression and Chromatin Accessibility using scRNA-Seq and scATAC-Seq. J Vis Exp. 10.3791/62239
  • Hoang and Wang et al. 2020. Gene regulatory networks controlling vertebrate retinal regeneration. Science. 10.1126/science.abb8598
  • Clark et al. 2019. Single-Cell RNA-Seq Analysis of Retinal Development Identifies NFI Factors as Regulating Mitotic Exit and Late-Born Cell Specification. Neuron.  10.1016/j.neuron.2019.04.010